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PulmiORA

Targeting COPD and Lung Fibrosis at the Molecular Core

A revolutionary first-in-class therapy that addresses the root cause of respiratory diseases, not just the symptoms.

COPD and Lung Fibrosis

Lung Fibrosis

What is Lung Fibrosis?

Lung fibrosis is a progressive condition characterized by irreversible scarring of lung tissue, leading to reduced lung elasticity and impaired oxygen exchange. As fibrosis advances, patients experience worsening breathlessness, chronic cough, fatigue, and declining functional capacity—often progressing to respiratory failure.

Despite available antifibrotic therapies, no current treatment reverses established fibrosis, leaving a critical gap for disease-modifying solutions.

What is COPD?

Chronic Obstructive Pulmonary Disease (COPD) is a chronic, progressive respiratory disease marked by persistent airflow limitation, typically caused by long-term inflammation and structural damage to the lungs. It includes chronic bronchitis and emphysema and is a leading cause of morbidity and mortality worldwide.

Patients suffer from breathlessness, frequent exacerbations, reduced exercise tolerance, and declining quality of life.

COPD Overview Diagram

Shared and Disease-Specific Drivers

Understanding the causes and risk factors is essential for developing targeted therapies that address both conditions.

Lung Fibrosis — Key Causes

  • Post-infectious lung injury — bacterial, viral, fungal pneumonia
  • Environmental and occupational exposure — silica, asbestos, industrial dust
  • Autoimmune diseases — rheumatoid arthritis, systemic sclerosis
  • Drug- and radiation-induced lung toxicity
  • Idiopathic origins, including IPF

COPD — Key Causes

  • Long-term tobacco smoking
  • Indoor and outdoor air pollution
  • Occupational exposure to fumes and chemicals
  • Recurrent lower respiratory infections
  • Genetic susceptibility (e.g., alpha-1 antitrypsin deficiency)

Shared Risk Factors

  • Advancing age
  • Chronic inflammatory burden
  • Repeated respiratory infections
  • Long-term exposure to airborne pollutants
  • Socioeconomic and environmental determinants of health

These overlapping drivers create a large, interconnected patient population, increasing the strategic relevance of therapies that address both airflow limitation and progressive lung damage.

Available Treatments for COPD and Lung Fibrosis

Current therapies provide symptom relief and slow progression but do not reverse established disease.

Lung Fibrosis Treatment

  • Approved antifibrotics: Pirfenidone, Nintedanib — slow progression but do not reverse scarring
  • Supportive care: Oxygen therapy, pulmonary rehabilitation, vaccination
  • Advanced options: Lung transplantation for eligible end-stage patients
  • Pipeline focus: Anti-fibrotic, anti-inflammatory, and regenerative therapies

COPD Treatment

  • Bronchodilators: LABA, LAMA
  • Anti-inflammatory therapy: Inhaled corticosteroids
  • Supportive care: Pulmonary rehabilitation, oxygen therapy
  • Advanced interventions: Non-invasive ventilation, lung volume reduction, transplant in select cases
  • Ongoing unmet need: Therapies that reduce disease progression, exacerbations, and structural lung damage

COPD and Lung Fibrosis Worldwide

Together, COPD and lung fibrosis affect tens of millions of patients worldwide and account for a substantial proportion of respiratory-related mortality and disability.

Global Impact

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High Morbidity

Chronic breathlessness, reduced quality of life, long-term oxygen dependence

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High Mortality

Progressive disease courses with limited curative options

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Major Economic Burden

Long-term therapy, frequent hospitalizations, productivity loss

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Rising Prevalence

Driven by aging populations, pollution, smoking, and post-infectious lung injury

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Global Scale

COPD affects over ~390 Million People Worldwide, with 3.5 Million Annual Deaths

This growing burden underscores the urgency for next-generation respiratory therapeutics.

Understanding Respiratory Disease at the Molecular Level

How COPD and Lung Fibrosis develop and progress through molecular pathways in the respiratory system

Disease Mechanism

Molecular Pathogenesis

Both COPD and Lung Fibrosis develop through complex molecular mechanisms involving the respiratory system. The disease progression is driven by chronic inflammation and structural changes at the cellular level. Viral, bacterial, and fungal pathogens interact with the respiratory epithelium, triggering inflammatory cascades. These pathogens affect RNA mechanisms and cellular processes, leading to persistent airway inflammation, mucus hypersecretion, progressive fibrosis, and destruction of lung tissue. Understanding these fundamental molecular pathways is crucial for developing targeted therapies that address the root causes rather than just managing symptoms.

Understanding the Respiratory Disease Crisis

COPD and Lung Fibrosis together affect hundreds of millions of people worldwide, causing millions of deaths annually. Current treatments only provide temporary symptom relief without addressing the underlying pathology.

Limited Treatment Options

Existing therapies like bronchodilators, corticosteroids, and antifibrotics provide only temporary symptom relief without addressing the root cause of disease progression.

Severe Side Effects

Current medications including steroidal medicines and antibiotics often come with severe long-term side effects, significantly impacting patient quality of life.

Disease Progression

Traditional treatments fail to halt or reverse disease progression, leading to continued lung function deterioration and irreversible tissue damage.

Growing Global Burden

Cases are projected to increase significantly through 2050 as populations age and environmental factors persist, creating an urgent need for breakthrough therapies.

Discover Our Revolutionary Solution

Learn how PulmiORA is transforming respiratory disease treatment by targeting COPD and Lung Fibrosis at their molecular core

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